1) What is the underlying physiology of “pain”?
Detailed Answer:
The kind of pressure experienced by a “touch” activates nociceptors (“pain” receptors). However, we don’t experience it as pain because of various mechanisms in pain modulation. Instead, we just feel it. If you put on comfortable clothes, you feel them initially but the primary nociceptive cutaneous fibers quickly desensitize and after a bit you can’t even feel that you’re wearing anything. Tapping someone, on the other hand (tonic pressure) activates nociceptive pathways that mechanical stimuli do not. Thus “pressure” can activate both the nociceptors that stabbing can and those that it cannot. A belt that is stiff, tight, and covering so much area is easily a noxious stimuli that becomes more painful as time goes on (imagine carrying a heavy bag- as you move it impedes your movement via the same mechanisms that make it uncomfortable to start with and coming in contact with your skin in a non-continuous, semi-tonic manner).
Summary: The perception of “pain” can result from two different kinds of sensory experiences. One includes everything from stabbing to poking to chemical burns. The other includes only a kind of constant “pressure” like being tapped repeatedly or wearing an uncomfortable belt for a long time. An uncomfortable belt can be experienced as the kind of pain that stabbing is, but also as a kind stabbing can’t.
2) How can wearing a tight belt prevent one from experiencing being stabbed?
Detailed Answer:
The “silent nociceptors” that are sensitive only to tonic pressure are mostly found in joints, but they are also common to the viscera. Such deep tissue pain is less inclined to subside and is not localized the way cutaneous pain is. Even if a belt were not intense enough to activate visceral “silent nociceptors” they are in joints like the spine which is pressed right up against the belt. Being stabbed, on the other hand, can’t activate either visceral or any other deep tissue mechano-insensitive nociceptors as it is a mechanical stimulus. The same nociceptive fields that are activated and modulated by the belt are those that the knife affects. If one were to put on such a belt after experiencing tissue trauma to some region the belt covers, one would feel increased pain (as hyperalgesia accompanies tissue damage). However, the same modulation processes that occur from wearing a belt that 1) is noxious just by being too tight and 2) is not applied continuously thanks to motion (making modulation more complex) and finally that is activating mechano-insensitive nociceptors will require a much more noxious mechanical stimulus to experience any sensation. Stabbing can do this through tissue damage if it e.g., damages afferent neuronal axons, but like everything in living systems and particularly within neurophysiology we are dealing with complex networks interacting in complex ways. Simplistically, the pain pathways that would be affected by tissue damage-induced hyperalgesia are already active, so indirect activation of peripheral afferent nociceptors STILL has to be transmitted through the CNS. As the modulation or changing of responses to afferent neurons begins at the spine, and the deep tissue nociceptors in the spine itself can be activated by the belt, it becomes a lot harder for a local, peripheral noxious stimulus to be experienced as such EVEN if it is experienced. Add to that the general effect of constricting blood flow, the behavioral influences on pain networks, a general arousal a recluse who receives constant death threats might experience setting foot in a public setting, etc., and you have a great combination of processes influencing the ways in which sensation is possible in general let one in an area covered by the belt. The main mechanism would be through peripheral nerve damage, but that’s exactly what a thin, fine blade avoids.
Summary:
If you’ve been carrying a heavy bag filled with bricks while jogging that continues to poke